- .Research progress of artificial blood:Basic & Clinical Medicine
- .Proteomics Study of Benzene Metabolite Hydroquinone Induced Hematotoxicity in K562 Cells:Biomedical and Environmental Sciences
- .The role of catechol-O-methyltransferase in catechol-enhanced erythroid differentiation of K562 cells:Toxicology and Applied Pharmacology
- .Phenolic Metabolites of Benzene Induced Caspase-Dependent Cytotoxicities to K562 Cells Accompanied with Decrease in Cell Surface Sialic Acids:Environmental Toxicology
- .Long-term exposure of K562 cells to benzene metabolites inhibited erythroid differentiation and elevated methylation in erythroid specific genes:Toxicology Research
- .5-氮杂-2’-脱氧胞苷对苯代谢物抑制K562细胞转铁蛋白受体表达的干预作用:毒理学杂志
- .The role of ROS in hydroquinone-induced inhibition of K562 cell erythroid differentiation:Biomedical and Environmental Sciences
- .吞蛋白A2在非网格蛋白内吞中的作用研究进展:生物化学与生物物理进展
- .Protease sensitivity and redistribution of CD71 and glycophorin A on K562 cells:Cellular and Molecular Biology
- .Changes in DNA methylation of erythroid-specific genes in K562 cells exposed to catechol in long term:Toxicology in Vitro

